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Post by beebs on May 1, 2011 3:56:54 GMT -5
None of these will bring instant relief, there is no magic bullet. It will help slow down the process, damage control effect. Caution: start on low dose, and self monitor. Rubbing hydrogen peroxide diluted at 3% or less, mix with water rub on sore joints. I read about SODzyme as injections, but I don't know how effective. (should be on Pubmed) Also read about Hyaluronic Acid, injections, but not much is known about that either. Some companies are selling this in tab form, (see further down). HA can be found in tabs forms too. Drink daily chicken broths and eat plenty of chicken cartilage. Warm Castor oil packs, Sesame Oil (warm), Oil of Oregano rub on sore joints. NAG ( N-Acetyl Glucosamine) + A, C, E, D, Boron, Fish Oils, GLA, rutin, biotin, quercetin, hesperidin, ginkgo (Ginkgo biloba), milk thistle (Silybum marianum) and proanthocyanidins. Bromelain, improvement was shown more than when taking it orally). MSM, Quercetin, EGCG (from Green Tea). Proboitics. Serrapeptase and a product called Boluoke, www.naturodoc.com/library/heart/boluoke.htm similar to Serrapeptase. Both are proteolytic helpful to reduce excess fibrin and athero, an as anti inflammatories!! (not meant as medical advice, passing on info)
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Post by pierce on May 2, 2011 6:36:26 GMT -5
I take NAG ( N-Acetyl Glucosamine) regularly. Three caps a day. If a person has problems with lectins, then this is especially useful. It is not a cure-all, but when I take at least 3 a day, I don't tend to need as much ibuprofen.
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Post by myrnawearshats on May 15, 2011 17:48:39 GMT -5
Are you guys seeing practitioners who specialize in supplements to decide on courses of treatment or just making it up as you go along?
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Post by mandysss on Dec 7, 2011 3:22:32 GMT -5
Hi,
I'm just reading some of these and feel I should point out that if you take Hyaluronic Acid you need to make sure that they check it for pharma impurities as it may be contaminated with fluoroquinolones.
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Post by beebs on Jul 12, 2012 14:54:43 GMT -5
I heard about Plasma Rich Therapy a while back. Excerpt "PRP therapy offers a promising solution to accelerate healing of tendon injuries and osteoarthritis naturally without subjecting the patient to significant risk. PRP is an emerging treatment in a new health sector known as ”Orthobiologics.” The philosophy is to merge cutting edge technology with the body’s natural ability to heal itself. Blood is made of RBC (Red Blood Cells), WBC (White Blood Cells), Plasma, and Platelets. When in their resting state, platelets look like sea sponges and when activated form branches. Platelets were initially known to be responsible for blood clotting. In the last 20 years we have learned that when activated in the body, platelets release healing proteins called growth factors. There are many growth factors with varying responsibilities, however cumulatively they accelerate tissue and wound healing. Therefore after increasing the baseline concentration of these platelets, we are able to deliver a powerful cocktail of growth factors that can dramatically enhance tissue recovery." www.orthohealing.com/plateletrichplasmatherapy-prp/
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Post by beebs on Sept 30, 2012 12:36:05 GMT -5
Article discusses the use of Cat's Claw, Maca, Boswellia and Leucine, which deactivates nuclear factor (NF)-kb chondrocyes. Concluded as preventing further damage by NO, iNos, MMP-9 and MMP-13 and switching catabolic gene expression to anabolic, to rejuvenate. The study dated 2011 mentions a product which was not yet commercially available at the time of this study, by Vital -g-Netics. \ Not mentioned in artricle, similar products used by Ayurvedic Practitioners, daily bone soups, chicken legs and feet which contains hyaluronic acid, silica, foods high Vit C for collagen, etc.. Effect of a Herbal-Leucine mix on the IL-1β-induced cartilage degradation and inflammatory gene expression in human chondrocytesNahid Akhtar1, Mark JS Miller2 and Tariq M Haqqi1* * Corresponding author: Tariq M Haqqi thaqqi@metrohealth.org Author Affiliations 1 Department of Medicine/Rheumatology, MetroHealth Medical Center, Case Western Reserve University, 2500 Metro Health Drive, Cleveland, Ohio 44109, USA Abstract Background Conventional treatments for the articular diseases are often effective for symptom relief, but can also cause significant side effects and do not slow the progression of the disease. Several natural substances have been shown to be effective at relieving the symptoms of osteoarthritis (OA), and preliminary evidence suggests that some of these compounds may exert a favorable influence on the course of the disease. The objective of this study was to investigate the anti-inflammatory/chondroprotective potential of a Herbal and amino acid mixture containing extract of the Uncaria tomentosa, Boswellia spp., Lepidium meyenii and L-Leucine on the IL-1β-induced production of nitric oxide (NO), glycosaminoglycan (GAG), matrix metalloproteinases (MMPs), aggrecan (ACAN) and type II collagen (COL2A1) in human OA chondrocytes and OA cartilage explants. Cont/.... www.ncbi.nlm.nih.gov/pubmed/9858130
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Post by beebs on Oct 4, 2012 15:54:36 GMT -5
Eating a bowl of cherries is helpful towards reducing high uric acid, low purine diet, and other lifestyle modification. Excerpt: "Risk for gout attacks was reduced by 75% when cherry intake was combined with allopurinol use. Dr. Zhang said, "We found that if subjects took allopurinol alone, it reduced the risk of gout attack by 53%; if subjects took cherry alone, it reduced the risk by 32%; if they took both, the risk of gout attack was reduced by 75%." These associations were discovered in a case-crossover study of 633 individuals with physician-diagnosed gout who were prospectively recruited and followed online for 1 year. When a participant reported a gout attack, the researchers asked about the onset date of the gout attack, symptoms and signs, medications, and potential risk factors (including daily intake of cherries and cherry extract) during the 2 days before the attack. Patients served as their own controls, so the same information was assessed for 2-day control periods not associated with gout attacks. A cherry serving was defined as one-half cup or 10 to 12 cherries." Article: www.medscape.com/viewarticle/772073?src=nldne
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Post by beebs on Dec 17, 2012 15:53:13 GMT -5
Before taking anything, consult your physician, watch out for side effects. Keep safe. Abstract (provisional) Background WIN-34B is a novel Oriental medicine, which represents the n-butanol fraction prepared from dried flowers of Lonicera japonica Thunb and dried roots of Anemarrhena asphodeloides BUNGE. The component herb of WIN-34B is used for arthritis treatment in East Asian countries. The aim of this study was to determine the cartilage-protective effects and mechanisms of WIN-34B and its major phenolic compounds, chlorogenic acid and mangiferin, in osteoarthritis (OA) human cartilage explants culture and chondrocytes. Methods The investigation focused on whether WIN-34B and its standard compounds protected cartilage in interleukin (IL)-1beta-stimulated cartilage explants culture and chondrocytes derived from OA patients. Also, the mechanisms of WIN-34B on matrix metalloproteinases (MMPs), tissue inhibitor of matrix metalloproteinases (TIMPs), inflammatory mediators, and mitogen-activated protein kinases (MAPKs) pathways were assessed. Results WIN-34B was not cytotoxic to cultured cartilage explants or chondrocytes. WIN-34B dose-dependently inhibited the release of glycosaminoglycan and type II collagen, increased the mRNA expression of aggrecan and type II collagen, and recovered the intensity of proteoglycan and collagen by histological analysis in IL-1beta-stimulated human cartilage explants culture. The cartilage protective effect of WIN-34B was similar to or better than that of chlorogenic acid and mangiferin. Compared to chlorogenic acid and mangiferin, WIN-34B displayed equal or greater decreases in the levels of MMP-1, MMP-3, MMP-13, ADAMTS-4, and ADAMTS-5, and markedly up-regulated TIMP-1 and TIMP-3. WIN-34B inhibited inflammatory mediators involved in cartilage destruction, such as prostaglandin E2, nitric oxide, tumor necrosis factor-alpha, and IL-1beta. The phosphorylation of extracellular signal-regulated kinase, c-Jun N-terminal kinase (JNK), and p38 was significantly reduced by WIN-34B treatment, while phosphorylation of JNK was only inhibited by chlorogenic acid or mangiferin in IL-1beta-stimulated chondrocytes. Conclusions WIN-34B is potentially valuable as a treatment for OA by virtue of its suppression of MMPs, ADAMTSs, and inflammatory mediators, and it's up-regulation of TIMP-1 and TIMP-3 involved in the MAPK pathway. de.mc1331.mail.yahoo.com/mc/welcome?.gx=1&.tm=1355689574&.rand=f4v3u3ou5jjfv#_pg=showMessage&sMid=0&fid=%2540S%2540Search&filterBy=&squery=biomed%20central&vp=1&.rand=889593378&midIndex=0&mid=2_5_22_1_161244039_ALxUfbwAAW7WUM6eqgijpgpKNsE&fromId=info@biomedcentral.com&clean&m=2_5_22_1_161244039_ALxUfbwAAW7WUM6eqgijpgpKNsE,2_5_1123_1_161051268_ALxUfbwAAXWHUM3PUw7J33Ir%2Bqk,2_5_2365_1_160810084_AL5UfbwAAVYAUMx0EwvzCmnpEGk,&sort=date&order=&startMid=0&.jsrand=6964523
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Post by beebs on Dec 29, 2012 12:27:58 GMT -5
Ayurvedic practitioners have been using night jasmine thousands of years to treat musculoskeletal issues, funal infections and other infections, and liver. Biodiversity of India, China and the Amazon (the latter holds more than one third of all species on the planet, India about 250,000 species of plants - stand corrected, can't remember exact figures), yields yet untapped sources for medicinal plants. The article shows success with reduction of pro-inflammatory markers such as cytokines after prolonged usage. Significant lessening of "necrosis factor-alpha, interleukin-1beta, and interleukin-6" were conclusive. Article: www.ncbi.nlm.nih.gov/pubmed?term=phytopherapy%20inflammed%20joints
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