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Post by beebs on May 6, 2011 6:00:52 GMT -5
I started taking ginger about two weeks ago, everyday. Either sliced in hot water with dried limes, or on its own, sometimes crushed with fresh organic carrots. The benefits are obvious since a recent D Dimer test show fibrin and coagulation optimal level. Cognition seems to improve a little, digestive system, hypoglycamic attacks and more... A new thread on CNS & Digestive System articles, they are intrinsically linked. It could also go under Phytopherapy. Essentially, don't excpect immediate results! Takes a while to work, if this can be seen as Epigenetics!! Compounds in Ginger and Magnolia bark (look up compounds for both) helpful for both the digestive system and CNS. Ayurvedic and TCM medicine have been using it for thousands of years successfully, I am not going to wait 30 years or more for Allopathic medicine to validate. I am using it now!! Reserch paper: www.zhion.com/herb/Ginger.htmlMagnolia bark, also anxyolitic, increases acethycholine, alleviates mensturations pains, balances sugar spikes, cortisol level, www.ayurvediccure.com/health/magnolia-bark-wonder-remedy/Ginger benefits: antimicrobial,anti fungal and more. Candida, E coli etc.. monosodium-glutamate toxicity, inhibits tumor growth (anti carcinogenic), gastric such as peptic ulcers, dyspepsia, h.pylori, androgenic (increases testosterones, increases libido, etc. relives nausea, thins blood, my (recent D Dimer test show amazing lowering of the fibrin and definitely less the coagulation) lowers cholesterol, anti-arrythmic (palpitations), anti diabetic, anti hypoglycemic, Anxyolitic (anxiety), Anti inflammatory for joints, cartilage etc. Powerful free radicals scavenger, and definitely decreases Nitric Oxide (very toxic), Neuron loss, scavenges neurotoxins, and neurodegenerative linked to CNS. "The combined treatment of honokiol and magnolol with ginger essential oil exerted synergistic antidepressant-like effects by prossibly regulating the serotonergic and gastroenteric system functions."
Read side effects: Finally, ginger may interact with surgical medications including anesthesia, leading to arrhythmias, poor wound healing, bleeding, photosensitivity reaction, and prolonged sedation. [28] Ginger has may also interact with certain anticoagulants and analgesics to cause bleeding. [29]
Also, see the post here about the health-quest.proboards.com/index.cgi?action=display&board=environmental&thread=12&page=1 and see the Enteric/Brain connection: www.psyking.net/id36.htm
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Post by piglet on May 6, 2011 9:31:54 GMT -5
Thank you Beebs for posting this. How much should one take? Is a tsp of fresh raw ginger in a cup of hot water enough?
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Post by beebs on May 6, 2011 13:11:05 GMT -5
Thank you Beebs for posting this. How much should one take? Is a tsp of fresh raw ginger in a cup of hot water enough? I am crushing fresh ginger and eating it, sometimes mixed with fresh carrots (also, crushed). Other times, slices of fresh ginger in warm water (just before boiling), or it will kill the good compounds. You can put dried or fresh limes/lemons in it too. Add honey if you can tolerate.
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Post by beebs on Sept 14, 2012 5:28:05 GMT -5
Main stream medicine catching up with brain and gut connection. MRIs do not usually show microscopic damage to brain.
Brain Abnormalities Common in Celiac Disease Patients Pam Harrison Authors and Disclosures
September 10, 2012 — Patients with celiac disease and neurologic symptoms have substantial structural and functional brain deficits as detected on magnetic resonance imaging (MRI), British investigators have observed.
Stuart Currie, PhD, and colleagues from the University of Sheffield, United Kingdom, found that patients with celiac disease and balance disturbances had significantly smaller cerebellar volumes than age- and sex-matched controls.
The same imaging studies showed significantly less grey matter density in multiple regions of the brain, including the cerebellum, among patients with celiac disease than among controls. Similarly, white matter abnormalities (WMAs) were identified in a significant proportion of the celiac disease cohort versus none of the controls.
"We were not surprised by the differences in cerebellar volume because we know that ataxia is one of the commonest neurological manifestations of celiac disease and we were not particularly surprised by the findings of WMAs either because we had previously described this entity of gluten encephalopathy — headache and WMAs — in patients with celiac disease so here, we just demonstrated that these abnormalities are common," co-investigator Marios Hadjivassiliou, MD, also from the University of Sheffield, told Medscape Medical News.
On the other hand, the fact that other areas of the brain are involved is interesting, he added, "in that they could potentially explain some of the other neurological problems such as sensory disturbances in patients who do not have peripheral neuropathy."
They might also help explain the anxiety that patients with celiac disease often have as well, he noted.
The study was published online August 20 in the Journal of Neurology, Neurosurgery and Psychiatry.
Neurologic Opinion
As the authors note, 33 eligible patients with celiac disease, mean age 44 years, were referred for neurologic consultation between January 2009 and March 2011.
Patients were referred for imaging for balance disturbances, headache, or sensory loss. The median time from the diagnosis of celiac disease to MRI was 2.1 years.
MRI findings revealed that cerebellar volumes were significantly smaller than those in controls, but only in patients who reported balance disturbances: 6.9% vs 7.4%, respectively (P < .05).
On the other hand, the volume of grey matter in both superior cerebellar hemispheres and in multiple cortical regions was significantly smaller in the patient group overall than in age- and sex-matched controls (P < .05).
Thirty-six percent of patients with celiac disease were also found to harbor WMAs. The prevalence of WMAs was greatest in the headache subgroup, the authors note.
Table. WMAs Among Patient Subgroups
Patient Subgroup Mean Age (y) Patients With WMA, n (%) Balance disturbances (n = 20) 52 6 (30) Headache (n = 6) 54 4 (67) Sensory loss (n = 4) 56 1 (25)
Gluten-Free Diet
As the authors discuss, two thirds of the patient group indicated they followed a gluten-free diet and were compliant with the diet when the imaging took place. The presence of WMAs in patients who appeared to have no known vascular risk factors implies a possible causal link between gluten sensitivity and WMAs, they add.
"What the findings mean is that patients with established celiac disease already on diet appear to have abnormal imaging that could account for some of their ongoing neurological complaints," Dr. Hadjivassiliou said.
"What it also means is that the gluten-free diet in these cases needs to be strict as exposure to small amounts of gluten may still be enough to continue the neurological insult. This is why close monitoring and demonstration that gluten-related antibodies are eliminated from the blood are essential."
Anna DePold Hohler, MD, from Boston University School of Medicine in Massachusetts told Medscape Medical News that the findings of decreased brain volume in patients with celiac disease help to solidify the neurologic implications of this disease.
"It also encourages further study in this area to better clarify the connection between the two and to eventually determine if treatment of celiac might also impact the brain," she added.
The precise pathologic mechanism between brain abnormalities and celiac disease is under investigation but it will probably implicate an immunologic reaction against the TG6 antigen, which is found in the brain.
The TG6 antigen shares 65% homology with TG2, the autoantigen in celiac disease, and TG3, the autoantigen in dermatitis herpetiformis.
The authors and Dr. DePold Hohler have disclosed no relevant financial relationships.
J Neurol Neurosurg Psychiatry. Published online August 20, 2012. Abstract.
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Post by Admin on Jul 20, 2015 4:07:39 GMT -5
Interesting study. Blame shiften to gluten for neuropsychiatric manifestations, could it not be related to transgenic foods, glyphosate insecticide etc ? Nutrients. 2015 Jul 8;7(7):5532-5539. Gluten Psychosis: Confirmation of a New Clinical Entity.Lionetti E1, Leonardi S2, Franzonello C3, Mancardi M4, Ruggieri M5, Catassi C6,7. Author information Abstract Non-celiac gluten sensitivity (NCGS) is a syndrome diagnosed in patients with symptoms that respond to removal of gluten from the diet, after celiac disease and wheat allergy have been excluded. NCGS has been related to neuro-psychiatric disorders, such as autism, schizophrenia and depression. A singular report of NCGS presenting with hallucinations has been described in an adult patient. We report a pediatric case of a psychotic disorder clearly related to NCGS and investigate the causes by a review of literature. The pathogenesis of neuro-psychiatric manifestations of NCGS is unclear. It has been hypothesized that: (a) a "leaky gut" allows some gluten peptides to cross the intestinal membrane and the blood brain barrier, affecting the endogenous opiate system and neurotransmission; or (b) gluten peptides may set up an innate immune response in the brain similar to that described in the gut mucosa, causing exposure from neuronal cells of a transglutaminase primarily expressed in the brain. The present case-report confirms that psychosis may be a manifestation of NCGS, and may also involve children; the diagnosis is difficult with many cases remaining undiagnosed. Well-designed prospective studies are needed to establish the real role of gluten as a triggering factor in neuro-psychiatric disorders. www.ncbi.nlm.nih.gov/pubmed/26184290
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