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Post by piglet on May 6, 2011 0:12:09 GMT -5
Digestive problems are very common with toxic reactions to pharmaceutical drugs. Personally I suffer from a total lack of appetite, lack of digestive motility, lack of digestive acids, gas, diarrhea, yellow stool, oily stool. When I do eat, it feels as if the food just lays in my stomach and rots for many many hours or entire days.
Wonder if others have/had such issues and what, if anything besides time, they found helpful.
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Post by Admin on Oct 20, 2011 15:03:21 GMT -5
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Post by Deleted on Dec 30, 2011 0:15:21 GMT -5
To the Administrator, Am most grateful for the info in the three links here. Healing starts with understanding and will begin again healing in this area fortified by this info. Also glad to be reminded of good work of Michael McEvoy.
A much improved New Year to everyone here!!!
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Post by Deleted on Jan 1, 2012 23:46:06 GMT -5
I wish you all a good new year and that everyone will find for himself the right way for improvement of his/her health. Namid
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Post by beebs on Sept 5, 2012 15:42:26 GMT -5
I wrote about various diets and bone broths on a different thread. I am finding the digestive system, most difficult to get a grip on. Mainly, because of allergies and inability to metaolize most things. I would not try cat's claw at this point. Dietary antioxidants protect gut epithelial cells from oxidant-induced apoptosisMark JS Miller1*, Fausto M Angeles1, Brian K Reuter1, Paul Bobrowski2 and Manuel Sandoval1 Author Affiliations 1 Center for Cardiovascular Sciences, Albany Medical College, Albany, New York, USA 2 Rainforest Phytoceuticals, LLC, Delmar, New York, USA Abstract Background The potential of ascorbic acid and two botanical decoctions, green tea and cat's claw, to limit cell death in response to oxidants were evaluated in vitro. Methods Cultured human gastric epithelial cells (AGS) or murine small intestinal epithelial cells (IEC-18) were exposed to oxidants – DPPH (3 μM), H2O2 (50 μM), peroxynitrite (300 μM) – followed by incubation for 24 hours, with antioxidants (10 μg/ml) administered as a 1 hour pretreatment. Cell number (MTT assay) and death via apoptosis or necrosis (ELISA, LDH release) was determined. The direct interactions between antioxidants and DPPH (100 μM) or H2O2 (50 μM) were evaluated by spectroscopy. Results The decoctions did not interact with H2O2, but quenched DPPH although less effectively than vitamin C. In contrast, vitamin C was significantly less effective in protecting human gastric epithelial cells (AGS) from apoptosis induced by DPPH, peroxynitrite and H2O2 (P < 0.001). Green tea and cat's claw were equally protective against peroxynitrite and H2O2, but green tea was more effective than cat's claw in reducing DPPH-induced apoptosis (P < 0.01). Necrotic cell death was marginally evident at these low concentrations of peroxynitrite and H2O2, and was attenuated both by cat's claw and green tea (P < 0.01). In IEC-18 cells, all antioxidants were equally effective as anti-apoptotic agents. Conclusions These results indicate that dietary antioxidants can limit epithelial cell death in response to oxidant stress. In the case of green tea and cat's claw, the cytoprotective response exceed their inherent ability to interact with the injurious oxidant, suggestive of actions on intracellular pathways regulating cell death. Full article www.biomedcentral.com/1472-6882/1/11
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Post by beebs on Sept 14, 2012 10:10:00 GMT -5
To date, I have never met anyone treated with anti TNF therapy recover!! Watch out herbal or compounds in foods which act as TNF supressors. Will post about this under Immune System thread, Immune System. Some will cause cytokine storm. Review Nature Reviews Gastroenterology and Hepatology 9, 496-503 (September 2012) | doi:10.1038/nrgastro.2012.125 Paradoxical inflammation induced by anti-TNF agents in patients with IBDIsabelle Cleynen & Séverine Vermeire Abstract Anti-TNF antibodies have acquired a prominent place in the management of IBD (including Crohn's disease and ulcerative colitis), rheumatologic conditions (such as rheumatoid arthritis, ankylosing spondylitis and psoriatic arthritis) and psoriasis. They have a good safety profile, especially when contraindications such as demyelinating disease, active infections and/or abscesses are ruled out, and when necessary precautions to prevent reactivation of tuberculosis are taken. However, with increasing use of these agents, paradoxical adverse events have been reported. Some of these features are shared with the underlying disease for which these drugs are given, making management of these conditions challenging. For example, anti-TNF therapy is used for the treatment of psoriasis, but psoriasiform lesions are sometimes observed in patients receiving therapy. Similarly, anti-TNF therapy is used for the treatment of rheumatologic diseases, but arthralgias and arthritis are sometimes observed in patients receiving anti-TNF agents. We review the paradoxical inflammation induced by anti-TNF agents in patients with IBD, provide hypotheses for the occurrence of this paradoxical inflammation and give practical advice on how to manage these patients. www.nature.com/nrgastro/journal/v9/n9/full/nrgastro.2012.125.html
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Post by beebs on Oct 31, 2012 17:31:22 GMT -5
Years ago, I use to take fresh organic Slovene propolis for digestive issues. Delicious and effective. It contains other healing properties, such as antibacterial, antioxidative against ROS, fatty acids metabolism in diabetes, Tualang honey causes cancer cells apoptosis, Green propolis also helps with intracellular repair such as mitochondria, CYP40 etc... Propolis medicinal value vary according to local botanical environment. (Taking it for months regularly is effective unless allergic or intolerant). Avoid, &contraindicated for inflammed stomach lining and problems with mucous production. Laxative effects and mechanism of action of Brazilian green propolisMamoru Kakino, Hiroshi Izuta, Kazuhiro Tsuruma, Yoko Araki, Masamitsu Shimazawa, Kenji Ichihara and Hideaki Hara For all author emails, please log on. BMC Complementary and Alternative Medicine 2012, 12:192 doi:10.1186/1472-6882-12-192 Published: 22 October 2012 Abstract (provisional) Background Brazilian green propolis is reported to have wide range of biological properties including antibacterial, anti-inflammatory, anti-influenza, and antioxidant activities. In the digestive system, a protective effect of propolis on gastric ulcer has been reported, but a laxative effect has not yet been reported. We investigated the effect and the mechanism of action of water and ethanol extracts of Brazilian green propolis. Methods We examined the laxative effect of propolis on stool frequency by administering orally an ethanol extract of propolis (EEP) or a water extract of propolis (WEP) at 10, 50, 100, or 500 mg/kg to normal mice. We then investigated the effects of propolis using constipation model mice induced by two types of drugs, loperamide (a mu opioid receptor agonist) and clonidine (an alpha-2 adrenergic receptor agonist). We also investigated the effects of WEP on gastrointestinal transit and contractional tension of the ileum to uncover the mechanism of action of WEP. Results Treatment with WEP, but not with EEP, significantly increased the weight of stools (p<0.01 at 500 mg/kg). WEP treatment significantly restored stool frequency and stool weight in clonidine-induced constipation model mice, but not in loperamide-induced constipation model mice. WEP treatment did not affect gastro-intestinal transit, but significantly increased the contractional tension of the isolated ileum of guinea pigs. This increase was inhibited by an acetylcholine receptor antagonist (atropine), but not by a 5-HT receptor antagonist (GR113808). Conclusion These findings indicate that WEP has laxative effects both in normal mice and in clonidine-induced constipation model mice. The laxative effects of WEP might be mediated by increased contractional tension of the ileum exerted at least in part via activation of an acetylcholine receptor. www.biomedcentral.com/1472-6882/12/192/abstract
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Post by beebs on Nov 25, 2012 9:48:55 GMT -5
Title of article blames "Orange Juice" causal in dangerous "new" syndrome. Is it the orange juice, pear, apple, bananas and other foods? The blame should be put on chems in foods, GMO, meds & vaccines, causing digestive inflammation, gut flora imbalances, leaky gut, gene mutations etc. Commercial orange juice is far from healthy. Manufacturers squeeze orange juice, put in huge storing barrels, oxygen removed. Companies that produce perfume and cosmetics, lace the juice with flavors and other chems. The article goes on about a "new" syndrome, called Food protein-induced enterocolitis syndrome (FPIES). To date, scientists know little about allergies. Tests are unreliable, the theory based on allergic being IgE mediated, that is immunoglobulin response, remains unsolved. Often tests are normal, patients suffer dangerous allergic reaction. Allergists call these episodes "false allergies" and are clueless about its mechanism. The article blames inflammation of the GI tract for dangerous responses to Orange Juice, cites a 2 year old boy, who presented with severe vomiting, tachycardia and dehydration after drinking orange juice and recurrence. www.medscape.com/viewarticle/774848?src=nldne
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Post by Admin on Aug 29, 2013 18:38:32 GMT -5
Diverse cultures have fermented foods worldwide, to preserve and for digestive health, using millets, barley, oatmeal, etc. A Swedish company marketed a product without milk in early 1990s based on an oatmeal gruel with Lactobacillus plantarum. This product was used on rats and showed improvement in immune Function, liver function, mucosa of digestive system and its immune System, translocation, and digestive inflammation. Humans showed improvement in fibrogen level (good for Heart health), reduction in bloating and increase in can increase carboxylic acids in feces. (not mentioned in the article). The mechanism is unknown. Not mentioned in the abstrat, the increase of carboxylic acids in feces, which includes other acids such as propionic, acetic and butyric acid is beneficial. Butryic acid shows Evidence of proliferating mucosa, causing tumor cell apoptosis, increases blood flow, Etc. Overall, elevation of carboxylic acid is helpful for restoring healthy digestive Immune system and reduction in inflammation. www.ncbi.nlm.nih.gov/pubmed/11157345
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