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Post by beebs on Jun 15, 2012 14:16:00 GMT -5
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Post by beebs on Jun 30, 2012 15:25:55 GMT -5
A number of adrenals support on the market, herbs, bovine or porcine glandular support.
Avoid exercises, (if your adrenals are dysfunctional, mitochondrial dysfunction etc.), it will deplete adrenals further, deplete nutrition, leading to adrenals worsening. Mild walking is advised, or even less for some
The general consensus time frame for mild dysfunctional adrenals to heal, will take between eight to twelve months.
Cortisol Tests are unreliable, serum shows the amount of cortisol in blood, saliva shows tissue and urine - metabolites. Symptoms are more reliable, and correlates with thyroid.
Before addressing the thyroid, adrenals should be addressed, or will create further imbalances.
Powder Macca sprinkled daily on your food, or drink in a shake, pure cacao (raw) daily, high in magnesium and zinc, Cordyseps for the immune system (but too strong for us, we need MODULATORS), rhodiola and so forth.
Treating adrenals should includes overall hormones modulators, and other systems. The adrenals do not work in isolation.
Updates in regards to adrenals will be posted next week.
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Post by beebs on Apr 1, 2013 10:19:39 GMT -5
The article from a toxicology journal discusses adrenals affected by medication. It explains in part the over active sympathetic nervous system and catecholamines surges, often misinterpreted as "panic attacks" or "stress" induced. Yes, physiological stress by meds toxicity. Toxicol Pathol. 2001 Jan-Feb;29(1):41-8. Adrenal gland: structure, function, and mechanisms of toxicity.Rosol TJ, Yarrington JT, Latendresse J, Capen CC. Source The Ohio State University, Department of Veterinary Biosciences, Columbus 43210, USA. rosol.1@osu.edu Abstract The adrenal gland is one of the most common endocrine organs affected by chemically induced lesions. In the adrenal cortex, lesions are more frequent in the zona fasciculata and reticularis than in the zona glomerulosa. The adrenal cortex produces steroid hormones with a 17-carbon nucleus following a series of hydroxylation reactions that occur in the mitochondria and endoplasmic reticulum. Toxic agents for the adrenal cortex include short-chain aliphatic compounds, lipidosis inducers, amphiphilic compounds, natural and synthetic steroids, and chemicals that affect hydroxylation. Morphologic evaluation of cortical lesions provides insight into the sites of inhibition of steroidogenesis. The adrenal cortex response to injury is varied. Degeneration (vacuolar and granular), necrosis, and hemorrhage are common findings of acute injury. In contrast, chronic reparative processes are typically atrophy, fibrosis, and nodular hyperplasia. Chemically induced proliferative lesions are uncommon in the adrenal cortex. The adrenal medulla contains chromaffin cells (that produce epinephrine, norepinephrine, chromogranin, and neuropeptides) and ganglion cells. Proliferative lesions of the medulla are common in the rat and include diffuse or nodular hyperplasia and benign and malignant pheochromocytoma. Mechanisms of chromaffin cell proliferation in rats include excess growth hormone or prolactin, stimulation of cholinergic nerves, and diet-induced hypercalcemia. There often are species specificity and age dependence in the development of chemically induced adrenal lesions that should be considered when interpreting toxicity data.
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