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Post by beebs on Jun 14, 2011 17:01:32 GMT -5
I have been taking Cinnamon for a couple of months or so, as well fresh ginger. I will continue to do so along with the Qi Gong treatment, and will introduce other compounds from herbs, veggies and fruits as alleriges lessens. Here is an excellent article, which does not go far enough, since it will take a long time before its recognized for its curative and preventive therapeuties. Meanwhile, Cinnamon is being used to treat diabetes in TCM & Ayurvedic medicine for thousands of years successfully along with other compounds!! Cinnamon: potential role in the prevention of insulin resistance, metabolic syndrome, and type 2 diabetes.J Diabetes Sci Technol. 2010 May;4(3):685-93 Cinnamon: potential role in the prevention of insulin resistance, metabolic syndrome, and type 2 diabetes. J Diabetes Sci Technol. 2010 May;4(3):685-93 Authors: Qin B, Panickar KS, Anderson RA Metabolic syndrome is associated with insulin resistance, elevated glucose and lipids, inflammation, decreased antioxidant activity, increased weight gain, and increased glycation of proteins. Cinnamon has been shown to improve all of these variables in in vitro, animal, and/or human studies. In addition, cinnamon has been shown to alleviate factors associated with Alzheimer's disease by blocking and reversing tau formation in vitro and in ischemic stroke by blocking cell swelling. In vitro studies also show that components of cinnamon control angiogenesis associated with the proliferation of cancer cells. Human studies involving control subjects and subjects with metabolic syndrome, type 2 diabetes mellitus, and polycystic ovary syndrome all show beneficial effects of whole cinnamon and/or aqueous extracts of cinnamon on glucose, insulin, insulin sensitivity, lipids, antioxidant status, blood pressure, lean body mass, and gastric emptying. However, not all studies have shown positive effects of cinnamon, and type and amount of cinnamon, as well as the type of subjects and drugs subjects are taking, are likely to affect the response to cinnamon. In summary, components of cinnamon may be important in the alleviation and prevention of the signs and symptoms of metabolic syndrome, type 2 diabetes, and cardiovascular and related diseases. PMID: 20513336 [PubMed - indexed for MEDLINE]
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Post by beebs on Jul 31, 2011 12:16:03 GMT -5
Hypo/pher glycemia, diabetes and other metabolic disorders, some which are not even diagnose-Able, ;D I took some vits and supps, but stopped those a while ago. Reported previously, I am taking ginger, cinnamon and tamarind. I also found out of a natural way to ward off diabetes or treat used by Ayurvedic medicine. wikidiabetes.blogspot.com/2009/01/pterocarpus-marsupium-indian-kino.html There are other links worth looking at. A cup made of wood from the Pterocarpus marsupium tree. Put warm water and leave over night. Drink in the morning and evening or as advised. I am seeing a surge of usage in Ayurvedic compounds in the US and the UK, for the last few years on health forums. Also noticed many supps are now mixing Ayurvedic compounds with vits. Most people don't realize that a lot of those compounds are actually from Ayurvedic medicine. I will order one, and give it a whirl. Found this on e-bay, likely, cgi.ebay.com/Vijaysar-Pterocarpous-Marsupium-Diabetic-Wooden-Cup-/380260609023I will ask my Ayur doc for a reputable source. There is also the use of copper cups. Leaving water overnight and drinking the water from the copper cup. Not read up on that one. It does crop up on several sites about its benefits. Taken from Ayurvedic Medicine. Thousands of published research papers, various theories, and more. Few leads below. Do you own search. Vits, supps, herbs etc: www.raysahelian.com/diabetes.htmlMovement to reverse diabetes naturally: www.rdnmovement.com/Dr Joe endo's blog: enews.endocrinemetabolic.com/Ayurvedic: holisticonline.com/Remedies/Diabetes/diabetes_ayurveda.htmVits & Supps: www.integratedhealth.com/hpdspec/naturaldiabetesmedicine.htmlwww.lef.org/protocols/abstracts/abstr-043.htmlHerbal Treatment: naturalherbalcleanse.com/Herbal-Remedies/articles/20-effective-herbal-remedies-for-diabetes-to-maintain-healthy-blood-sugarTCM: tcmwell.com/TCMNaturalTherapy/TCMNaturalTh/Diabetes-mellitus-with-herbs-in-tcm.htmlResearch: illustrates the limitations of various contradictory theories about diabetes - www.ncbi.nlm.nih.gov/pmc/articles/PMC2780862/news.wustl.edu/news/Pages/3330.aspxwww.lef.org/offshoredrugs.htmjcem.endojournals.org/content/91/11/4657.fullwww.naturalnews.com/027398_chromium_diabetes_insulin.htmlwww.ehow.com/facts_6158630_gila-monster-venom-diabetes.htmllots of litigation using Byetta, high rate of strokes and heart attacks, but then, so does insulin!!
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Post by beebs on Dec 27, 2011 13:10:46 GMT -5
Good old Stevia, little did I know, it was being used in some countries to treat some metabolic syndrome. No wonder the FDA and BP violently opposed import of Stevia for over 30 years to the USA... Stevioside induces antihyperglycaemic, insulinotropic and glucagonostatic effects in vivo: studies in the diabetic Goto-Kakizaki (GK) rats.Jeppesen PB, Gregersen S, Alstrup KK, Hermansen K. Source Department of Endocrinology and Metabolism C, Aarhus University Hospital, Denmark. pbj@mail-telia.dk Abstract Extracts of leaves from the plant Stevia rebaudiana Bertoni have been used in the traditional treatment of diabetes in Paraguay and Brazil. Recently, we demonstrated a direct insulinotropic effect in isolated mouse islets and the clonal beta cell line INS-1 of the glycoside stevioside that is present in large quantity in these leaves. Type 2 diabetes is a chronic metabolic disorder that results from defects in both insulin and glucagon secretion as well as insulin action. In the present study we wanted to unravel if stevioside in vivo exerts an antihyperglycaemic effect in a nonobese animal model of type 2 diabetes. An i.v. glucose tolerance test (IVGT) was carried out with and without stevioside in the type 2 diabetic Goto-Kakizaki (GK) rat, as well as in the normal Wistar rat. Stevioside (0.2 g/kg BW) and D-glucose (2.0 g/kg BW) were administered as i.v. bolus injections in anaesthetized rats. Stevioside significantly suppressed the glucose response to the IVGT in GK rats (incremental area under the curve (IAUC): 648 +/- 50 (stevioside) vs 958 +/- 85 mM x 120 min (control); P < 0.05) and concomitantly increased the insulin response (IAUC: 51116 +/- 10967 (stevioside) vs 21548 +/- 3101 microU x 120 min (control); P < 0.05). Interestingly, the glucagon level was suppressed by stevioside during the IVGT, (total area under the curve (TAUC): 5720 +/- 922 (stevioside) vs 8713 +/- 901 pg/ml x 120 min (control); P < 0.05). In the normal Wistar rat stevioside enhanced insulin levels above basal during the IVGT (IAUC: 79913 +/- 3107 (stevioside) vs 17347 +/- 2882 microU x 120 min (control); P < 0.001), however, without altering the blood glucose response (IAUC: 416 +/- 43 (stevioside) vs 417 +/- 47 mM x 120 min (control)) or the glucagon levels (TAUC: 5493 +/- 527 (stevioside) vs 5033 +/- 264 pg/ml x 120 min (control)). In conclusion, stevioside exerts antihyperglycaemic, insulinotropic, and glucagonostatic actions in the type 2 diabetic GK rat, and may have the potential of becoming a new antidiabetic drug for use in type 2 diabetes. www.ncbi.nlm.nih.gov/pubmed/11924770
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Post by beebs on Jan 15, 2012 12:59:16 GMT -5
We have a long way before catching up with "folklore" medicine... The study is about bitter gourd. It tastes yukky...difficult to find. Wild bitter gourd improves metabolic syndrome: A preliminary dietary supplementation trialChung-Huang Tsai, Emily Chin-Fun Chen, Hsin-Sheng Tsay and Ching-jang Huang For all author emails, please log on. Nutrition Journal 2012, 11:4 doi:10.1186/1475-2891-11-4 Published: 13 January 2012 Abstract (provisional) Background Bitter gourd (Momordica charantia L.) is a common tropical vegetable that has been used in traditional or folk medicine to treat diabetes. Wild bitter gourd (WBG) ameliorated metabolic syndrome (MetS) in animal models. We aimed to preliminarily evaluate the effect of WBG supplementation on MetS in Taiwanese adults. Methods A preliminary open-label uncontrolled supplementation trial was conducted in eligible fulfilled the diagnosis of MetS from May 2008 to April 2009. A total of 42 eligible (21 men and 21 women) with a mean age of 45.7+/- 11.4 years (23 to 63 years) were supplemented with 4.8 gram lyophilized WBG powder in capsules daily for three months and were checked for MetS at enrollment and follow-up monthly. After supplementation was ceased, the participants were continually checked for MetS monthly over an additional three-month period. MetS incidence rate were analyzed using repeated-measures generalized linear mixed models according to the intention-to-treat principle. Results After adjusting for sex and age, the MetS incidence rate (standard error, p value) decreased by 7.1% (3.7%, 0.920), 9.5% (4.3%, 0.451), 19.0% (5.7%, 0.021), 16.7% (5.4%, 0.047), 11.9% (4.7%, 0.229) and 11.9% (4.7%, 0.229) at visit 2, 3, 4, 5, 6, and 7 compared to that at baseline (visit 1), respectively. The decrease in incidence rate was highest at the end of the three-month supplementation period and it was significantly different from that at baseline (p=0.021). The difference remained significant at end of the 4th month (one month after the cessation of supplementation) (p=0.047) but the effect diminished at the 5th and 6th months after baseline. The waist circumference also significantly decreased after the supplementation (p<0.05). The WBG supplementation was generally well-tolerated. Conclusion This is the first report to show that WBG improved MetS in human which provides a firm base for further randomized controlled trials to evaluate the efficacy of WBG supplementation. The complete article is available as a provisional PDF. The fully formatted PDF and HTML versions are in production. www.nutritionj.com/content/11/1/4/abstract
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Post by beebs on Jul 6, 2012 12:01:10 GMT -5
Hperinsulinemia: a recipe to alleviate symptoms + published research Look up other foods that contain inulin, important to regulate blood sugar and insulin level. These two overlap. Too much glucose or insulin will also contribute to brain inflammation. A home made juice that will control both: Mix beet juice, alfalfa and Jerusalem artichoke tuber (J Artichoke contains inulin (not insulin) which helps regulate insulin and glucose). Eur J Clin Nutr. 2011 Dec;65(12):1279-86. doi: 10.1038/ejcn.2011.116. Epub 2011 Jun 29. Inulin increases short-term markers for colonic fermentation similarly in healthy and hyperinsulinaemic humans.Fernandes J, Vogt J, Wolever TM. Source Department of Nutritional Sciences, Faculty of Medicine, University of Toronto, Toronto, Ontario, Canada. Abstract BACKGROUND/OBJECTIVES: Colonic fermentation of dietary fibre produces short-chain fatty acids (SCFAs), acetate, propionate and butyrate, which may protect against type 2 diabetes by reducing serum free-fatty acids (FFAs). Since hyperinsulinaemia is associated with insulin resistance and increased diabetes risk, the main objective was to compare markers of colonic fermentation after acute inulin ingestion in subjects with normal (<40 pmol/l, NI) and high (≥40 pmol/l, HI) plasma insulin. SUBJECTS/METHODS: Overnight fasted NI (n=9) and HI (n=9) subjects were studied for 4 h on two separate days after consuming 300 ml drinks containing 75 g glucose (Glucose) or 75 g glucose plus 24 g inulin (Inulin) using a randomized, single-blind, crossover design. RESULTS: Inulin elicited a higher breath hydrogen and methane areas under the curve (AUC), but the increases in SCFA responses were not statistically significant. Mean serum-acetate concentration over the 4-h study period was higher in NI than in HI subjects (44.3 ± 6.9 vs 22.5 ± 3.7 μmol/l, P=0.001). The rate of rebound of FFA was reduced by Inulin, with FFA at 4 h being less after Inulin than Glucose, regardless of insulin status (0.310 ± 0.028 vs 0.432 ± 0.042 mEq/l, P=0.008). CONCLUSIONS: This suggests that inulin increases short-term markers for colonic fermentation, but a longer study period may be necessary to observe differences in SCFA production. The reason for the lower serum acetate in HI is unclear but may be due to reduced absorption, increased clearance or decreased endogenous production. This suggests the need to compare acetate kinetics in normal and hyperinsulinaemic subjects. www.ncbi.nlm.nih.gov/pubmed/21712835Applications of inulin and oligofructose in health and nutrition. Kaur N, Gupta AK.Source Department of Biochemistry and Chemistry, Punjab Agricultural University, Ludhiana 141 004, India. nkaur@rediffmail.com Abstract Inulin and oligofructose belong to a class of carbohydrates known as fructans. The main sources of inulin and oligofructose that are used in the food industry are chicory and Jerusalem artichoke. Inulin and oligofructose are considered as functional food ingredients since they affect the physiological and biochemical processes in rats and human beings, resulting in better health and reduction in the risk of many diseases. Experimental studies have shown their use as bifidogenic agents, stimulating the immune system of the body, decreasing the pathogenic bacteria in the intestine, relieving constipation, decreasing the risk of osteoporosis by increasing mineral absorption, especially of calcium, reducing the risk of atherosclerosis by lowering the synthesis of triglycerides and fatty acids in the liver and decreasing their level in serum. These fructans modulate the hormonal level of insulin and glucagon, thereby regulating carbohydrate and lipid metabolism by lowering the blood glucose levels; they are also effective in lowering the blood urea and uric acid levels, thereby maintaining the nitrogen balance. Inulin and oligofructose also reduce the incidence of colon cancer. The biochemical basis of these beneficial effects of inulin and oligofructose have been discussed. Oligofructose are non cariogenic as they are not used by Streptococcus mutans to form acids and insoluble glucans that are the main culprits in dental caries. Because of the large number of health promoting functions of inulin and oligofructose, these have wide applications in various types of foods like confectionery, fruit preparations, milk desserts, yogurt and fresh cheese, baked goods, chocolate, ice cream and sauces. Inulin can also be used for the preparation of fructose syrups. www.ncbi.nlm.nih.gov/pubmed/12571376
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Post by beebs on Sept 29, 2012 16:55:47 GMT -5
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Post by Admin on Sept 30, 2012 12:55:46 GMT -5
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Post by beebs on Oct 7, 2012 5:24:19 GMT -5
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Post by Admin on Oct 16, 2012 15:02:38 GMT -5
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Post by beebs on Oct 31, 2012 18:44:26 GMT -5
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Post by beebs on Jan 12, 2013 9:59:08 GMT -5
Unani medicine is closely related to the Ayurvedic system. en.wikipedia.org/wiki/Medicine,_unani I first heard about it a few years ago, from a desperate parent looking for non toxic drugs treatment for his severely allergic child. Found this article which discusses a formulation - Qurs Tabasheer which contains Tukhme Khurfa (Portulaca oleracea seed), Gule Surkh (Rosa damascena flower), Gulnar (Punica granatum flower), Tabasheer (Bambusa arundinasia dried exudate on node), Tukhme Kahu (Lactuca sativa Linn seed), for diabetes treatment. The study involved inducing diabetes in wistar rates with Streptozotocin. Rats showed increase in markers for diabetes, tryglycerides and all cholesterol except lower HDL level. The study shows strong evidence of Qurs Tabasheer more beneficial than the usual Glimeperide for hypoglycemia. Study: www.biomedcentral.com/1472-6882/13/10/abstract
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Post by beebs on Apr 1, 2013 12:26:25 GMT -5
The article dates 2006, non wonder the study was halted, it would loose billions worth of revenue for Big Pharma. Ingrained belief about irreversible disease will remain unless thinking out of the box. All diseases are basically miscommunication between cells, its about restoring healthy cell functions. For some multisystemic long term chronic illness and accumulation of free radicals, blockages of all seven detoxification pathways etc..may be difficult to reverse.. TRPV1+ Sensory Neurons Control β Cell Stress and Islet Inflammation in Autoimmune DiabetesArticle has an altmetric score of 14 Rozita Razavi1, 2, 4, 7, Yin Chan1, 2, 4, 7, F. Nikoo Afifiyan1, 2, 4, 7, Xue Jun Liu1, 3, 7, Xiang Wan1, 3, Jason Yantha1, 2, 4, Hubert Tsui1, 2, 4, Lan Tang1, 2, 4, Sue Tsai5, Pere Santamaria5, John P. Driver5, David Serreze6, Michael W. Salter1, 3 and H.-Michael Dosch1, 2, 4, Go To Corresponding Author, 1 Neurosciences and Mental Health Program, The Hospital for Sick Children, Research Institute, University of Toronto, Toronto, ON, Canada, M5G 1X8 2 Department of Pediatrics, The Hospital for Sick Children, Research Institute, University of Toronto, Toronto, ON, Canada, M5G 1X8 3 Department of Physiology, The Hospital for Sick Children, Research Institute, University of Toronto, Toronto, ON, Canada, M5G 1X8 4 Department of Immunology, The Hospital for Sick Children, Research Institute, University of Toronto, Toronto, ON, Canada, M5G 1X8 5 Julia McFarlane Diabetes Research Centre and Department of Microbiology and Infectious Diseases, Faculty of Medicine, University of Calgary, Calgary, AB, Canada T2N 4N1 6 The Jackson Laboratory, Bar Harbor, ME 04609, USA Corresponding author 7 These authors contributed equally to the work. Summary In type 1 diabetes, T cell-mediated death of pancreatic β cells produces insulin deficiency. However, what attracts or restricts broadly autoreactive lymphocyte pools to the pancreas remains unclear. We report that TRPV1+ pancreatic sensory neurons control islet inflammation and insulin resistance. Eliminating these neurons in diabetes-prone NOD mice prevents insulitis and diabetes, despite systemic persistence of pathogenic T cell pools. Insulin resistance and β cell stress of prediabetic NOD mice are prevented when TRPV1+ neurons are eliminated. TRPV1NOD, localized to the Idd4.1 diabetes-risk locus, is a hypofunctional mutant, mediating depressed neurogenic inflammation. Delivering the neuropeptide substance P by intra-arterial injection into the NOD pancreas reverses abnormal insulin resistance, insulitis, and diabetes for weeks. Concordantly, insulin sensitivity is enhanced in trpv1−/− mice, whereas insulitis/diabetes-resistant NODxB6Idd4-congenic mice, carrying wild-type TRPV1, show restored TRPV1 function and insulin sensitivity. Our data uncover a fundamental role for insulin-responsive TRPV1+ sensory neurons in β cell function and diabetes pathoetiology. www.cell.com/abstract/S0092-8674%2806%2901465-6
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